Which medication is known to decrease inflammation and support the formation of fibrous caps to prevent plaque rupture?

Statins are widely recognized for their role in lipid-lowering therapy, but their benefits extend beyond cholesterol management. One significant aspect of statins is their anti-inflammatory properties. They help reduce systemic inflammation, which is crucial in the context of atherosclerosis, as inflammation plays a vital role in plaque formation and stability.

In addition to lowering low-density lipoprotein (LDL) cholesterol levels, statins promote plaque stabilization by supporting the formation of fibrous caps over atherosclerotic lesions. A strong fibrous cap is essential for preventing plaque rupture, which can lead to acute cardiovascular events such as myocardial infarction or stroke. By enhancing this fibrous cap formation and reducing the inflammation associated with plaque instability, statins effectively contribute to overall cardiovascular protection.

Other medications listed, such as beta-blockers, ACE inhibitors, and anticoagulants, serve different purposes in cardiovascular management but do not have the same dual role of reducing inflammation and stabilizing plaques as statins do.